Mutant of Polyoma Virus with Impaired Adsorption to BHK Cells
Author:
Affiliation:
1. Department of Pathology, New York University School of Medicine, New York, New York 10016
2. Department of Microbiology, University of Illinois Medical Center, Chicago, Illinois 60680
Abstract
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/jvi.13.4.931-934.1974
Reference12 articles.
1. Susceptibility of cells in different stages of the mitotic cycle to transformation by polyoma virus;Basillico C.;Virology,1966
2. Origin of the thymidine kinase induced by polyoma virus in productively infected cells;Basilico C.;J. Virol.,1969
3. Host range mutants of polyoma virus;Benjamin T. L.;Proc. Nat. Acad. Sci. U.S.A.,1970
4. Absence of a cell membrane alteration function in non-transforming mutants of polyoma virus;Benjamin T. L.;Proc. Nat. Acad. Sci. U.S.A.,1970
5. Some characteristics of large-plaque and small-plaque lines of polyoma virus;Diamond L.;Virology,1964
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1. A single-amino-acid substitution in polyomavirus VP1 correlates with plaque size and hemagglutination behavior;Journal of Virology;1991-01
2. Interactions of Animal Viruses with Cell Surface Receptors;The Receptors;1985
3. Sialyloligosaccharide receptors of binding variants of polyoma virus;Virology;1983-10
4. Differences in the subpopulations of the structural proteins of polyoma virions and capsids: biological functions of the multiple VP1 species;Journal of Virology;1981-01
5. Differential adsorption of polyoma virions and capsids to mouse kidney cells and guinea pig erythrocytes;Journal of Virology;1979-11
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