Parathyroid Hormone-Related Peptide Represses Chondrocyte Hypertrophy through a Protein Phosphatase 2A/Histone Deacetylase 4/MEF2 Pathway
Author:
Affiliation:
1. Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, 240 Longwood Ave., Boston, Massachusetts 02115
2. Department of Pharmacology & Cancer Biology, Duke University, Durham, North Carolina 27710
Abstract
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.00415-09
Reference36 articles.
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2. Arnold, M. A., Y. Kim, M. P. Czubryt, D. Phan, J. McAnally, X. Qi, J. M. Shelton, J. A. Richardson, R. Bassel-Duby, and E. N. Olson. 2007. MEF2C transcription factor controls chondrocyte hypertrophy and bone development. Dev. Cell12:377-389.
3. Backs, J., K. Song, S. Bezprozvannaya, S. Chang, and E. N. Olson. 2006. CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy. J. Clin. Investig.116:1853-1864.
4. Belfield, J. L., C. Whittaker, M. Z. Cader, and S. Chawla. 2006. Differential effects of Ca2+ and cAMP on transcription mediated by MEF2D and cAMP-response element-binding protein in hippocampal neurons. J. Biol. Chem.281:27724-27732.
5. Berdeaux, R., N. Goebel, L. Banaszynski, H. Takemori, T. Wandless, G. D. Shelton, and M. Montminy. 2007. SIK1 is a class II HDAC kinase that promotes survival of skeletal myocytes. Nat. Med.13:597-603.
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