Reply to Das et al., “TIM1 (HAVCR1): an Essential ‘Receptor’ or an ‘Accessory Attachment Factor’ for Hepatitis A Virus?”
Author:
Affiliation:
1. Center for Biologics Evaluation and Research, Food and Drug Administration, Silver Spring, Maryland, USA
Funder
HHS | NIH | National Institute of Allergy and Infectious Diseases
HHS | U.S. Food and Drug Administration
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.02040-18
Reference11 articles.
1. HAVCR1 (CD365) and Its Mouse Ortholog Are Functional Hepatitis A Virus (HAV) Cellular Receptors That Mediate HAV Infection
2. Identification of a surface glycoprotein on African green monkey kidney cells as a receptor for hepatitis A virus.
3. Polymorphisms of the Hepatitis A Virus Cellular Receptor 1 in African Green Monkey Kidney Cells Result in Antigenic Variants That Do Not React with Protective Monoclonal Antibody 190/4
4. The Human Homolog of HAVcr-1 Codes for a Hepatitis A Virus Cellular Receptor
5. The Cys-Rich Region of Hepatitis A Virus Cellular Receptor 1 Is Required for Binding of Hepatitis A Virus and Protective Monoclonal Antibody 190/4
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1. Grouper TIM-1 promotes nodavirus infection by inhibiting immune and inflammation response;Fish & Shellfish Immunology;2024-10
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