KatG as Counterselection Marker for Nontuberculous Mycobacteria
Author:
Affiliation:
1. Institute of Medical Microbiology, University of Zurich, Zurich, Switzerland
2. National Center for Mycobacteria, Zurich, Switzerland
Funder
University of Zurich
Georg and Bertha Schwyzer-Winiker Foundation
Foundation for Research in Science and the Humanities at the University of Zurich
Lungenliga Schweiz
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology
Link
https://journals.asm.org/doi/pdf/10.1128/AAC.02508-19
Reference25 articles.
1. Differential Sensitivity of Mycobacteria to Isoniazid Is Related to Differences in KatG-Mediated Enzymatic Activation of the Drug
2. The catalase—peroxidase gene and isoniazid resistance of Mycobacterium tuberculosis
3. Characterization of the katG gene encoding a catalase-peroxidase required for the isoniazid susceptibility of Mycobacterium tuberculosis
4. inhA , a Gene Encoding a Target for Isoniazid and Ethionamide in Mycobacterium tuberculosis
5. Isoniazid’s Bactericidal Activity Ceases because of the Emergence of Resistance, Not Depletion ofMycobacterium tuberculosisin the Log Phase of Growth
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