How the Bacterial Pathogen Listeria monocytogenes Mediates the Switch from Environmental Dr. Jekyll to Pathogenic Mr. Hyde
Author:
Affiliation:
1. Department of Food Science, Cornell University, Ithaca, New York
2. Seattle Biomedical Research Institute and the Departments of Pathobiology and Microbiology, University of Washington, Seattle, Washington
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.74.5.2505-2512.2006
Reference122 articles.
1. Bassler, B. L. 2002. Small talk. Cell-to-cell communication in bacteria. Cell109:421-424.
2. Beauregard, K. E., K.-D. Lee, R. J. Collier, and J. A. Swanson. 1997. pH-dependent perforation of macrophage phagosomes by listeriolysin O from Listeria monocytogenes. J. Exp. Med.186:1159-1163.
3. Identification of the Gene Encoding the Alternative Sigma Factor ς B from Listeria monocytogenes and Its Role in Osmotolerance
4. Contribution of Three Bile-Associated Loci, bsh , pva , and btlB , to Gastrointestinal Persistence and Bile Tolerance of Listeria monocytogenes
5. Regulation of hly Expression in Listeria monocytogenes by Carbon Sources and pH Occurs through Separate Mechanisms Mediated by PrfA
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