Different Erythromycin Resistance Mechanisms in Group C and Group G Streptococci-Reference-Cited by-同舟云学术

Different Erythromycin Resistance Mechanisms in Group C and Group G Streptococci

Published:1998-06 Issue:6 Volume:42 Page:1493-1494
ISSN:0066-4804
Container-title:Antimicrobial Agents and Chemotherapy
language:en
Short-container-title:Antimicrob Agents Chemother

Author:

Kataja Janne¹,Seppälä Helena¹²,Skurnik Mikael³,Sarkkinen Hannu⁴,Huovinen Pentti¹

Affiliation:

1. Antimicrobial Research Laboratory, National Public Health Institute,1

2. Department of Ophthalmology, University of Turku,2 and

3. Turku Centre for Biotechnology, University of Turku and Åbo Akademi University,3 Turku, and

4. Central Hospital of Päijät-Häme, Lahti,4 Finland

Abstract

ABSTRACT Different mechanisms of erythromycin resistance predominate in group C and G streptococcus (GCS and GGS, respectively) isolates collected from 1992 to 1995 in Finland. Of the 21 erythromycin-resistant GCS and 32 erythromycin-resistant GGS isolates, 95% had the mefA or mefE drug efflux gene and 94% had the ermTR methylase gene, respectively.

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Pharmacology (medical),Pharmacology

Link

https://journals.asm.org/doi/pdf/10.1128/AAC.42.6.1493

Reference20 articles.

1. Erythromycin-resistant beta-haemolytic streptococci in North Yorkshire.;Barnham M.;J. Infect.,1986

2. Cloning and hybridization analysis of ermP, a macrolide-lincosamide-streptogramin B resistance determinant from Clostridium perfringens

3. Molecular cloning and functional analysis of a novel macrolide-resistance determinant, mefA, from Streptococcus pyogenes.;Clancy J.;Mol. Microbiol.,1996

4. Kataja J. P. Huovinen M. Skurnik and H. Seppälä. Unpublished data.

5. Bacterial resistance to macrolide, lincosamide, and streptogramin antibiotics by target modification

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