Yersinia pestis Exploits Early Activation of MyD88 for Growth in the Lungs during Pneumonic Plague-Reference-Cited by-同舟云学术

Yersinia pestis Exploits Early Activation of MyD88 for Growth in the Lungs during Pneumonic Plague

Published:2019-04 Issue:4 Volume:87 Page:
ISSN:0019-9567
Container-title:Infection and Immunity
language:en
Short-container-title:Infect Immun

Author:

Olson Rachel M.¹²,Dhariwala Miqdad O.¹²,Mitchell William J.¹,Anderson Deborah M.¹²

Affiliation:

1. Department of Veterinary Pathobiology, University of Missouri, Columbia, Missouri, USA

2. Laboratory of Infectious Disease Research, University of Missouri, Columbia, Missouri, USA

Abstract

Yersinia pestis causes bubonic, pneumonic, and septicemic plague. Although no longer responsible for pandemic outbreaks, pneumonic plague continues to be a challenge for medical treatment and has been classified as a reemerging disease in some parts of the world.

Funder

NIH/NIAID

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Immunology,Microbiology,Parasitology

Link

https://journals.asm.org/doi/pdf/10.1128/IAI.00757-18

Reference37 articles.

1. IRAK (Pelle) Family Member IRAK-2 and MyD88 as Proximal Mediators of IL-1 Signaling

2. MyD88 Is an Adaptor Protein in the hToll/IL-1 Receptor Family Signaling Pathways

3. MyD88: a central player in innate immune signaling