Global Regulatory Pathways Converge To Control Expression of Pseudomonas aeruginosa Type IV Pili-Reference-Cited by-同舟云学术

Global Regulatory Pathways Converge To Control Expression of Pseudomonas aeruginosa Type IV Pili

Published:2022-02-22 Issue:1 Volume:13 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Coggan Kimberly A.¹,Higgs Matthew G.¹^ORCID,Brutinel Evan D.²,Marden Jeremiah N.³,Intile Peter J.²,Winther-Larsen Hanne C.⁴,Koomey Michael⁵,Yahr Timothy L.²^ORCID,Wolfgang Matthew C.¹³

Affiliation:

1. Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, North Carolina, USA

2. Department of Microbiology and Immunology, University of Iowa, Iowa City, Iowa, USA

3. Marsico Lung Institute, University of North Carolina, Chapel Hill, North Carolina, USA

4. Center for Integrative Microbial Evolution and Department of Pharmacology and Pharmaceutical Biosciences, University of Oslo, Oslo, Norway

5. Department of Biosciences, Section for Genetics and Evolutionary Biology and Centre for Ecological and Evolutionary Synthesis, University of Oslo, Oslo, Norway

Abstract

Pseudomonas aeruginosa is an opportunistic pathogen responsible for a wide range of infections. An extensive repertoire of virulence factors aid in P. aeruginosa pathogenesis.

Funder

Division of Intramural Research, National Institute of Allergy and Infectious Diseases

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mbio.03696-21

Reference67 articles.

1. Pseudomonas aeruginosa, mucoidy and the chronic infection phenotype in cystic fibrosis

2. Production of mucoid microcolonies by Pseudomonas aeruginosa within infected lungs in cystic fibrosis

3. Microbial pathogenesis in cystic fibrosis: mucoid Pseudomonas aeruginosa and Burkholderia cepacia.