Inhibition of Metalloprotease TRABD2A Facilitates the Study of HIV-1 Replication in Resting CD4 + T Cells-Reference-Cited by-同舟云学术

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Inhibition of Metalloprotease TRABD2A Facilitates the Study of HIV-1 Replication in Resting CD4 + T Cells

Published:2020-02-14 Issue:5 Volume:94 Page:
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:J Virol

Author:

Dong Jinxiu¹²,Liang Guoxin¹³⁴²,Shang Hong¹⁴²

Affiliation:

1. Key Laboratory of AIDS Immunology of Ministry of Health, Department of Laboratory Medicine, Shenyang, China

2. Key Laboratory of AIDS Immunology, Chinese Academy of Medical Sciences, Shenyang, China

3. Cancer Therapy Research Institute, Shenyang, China

4. National Clinical Research Center for Laboratory Medicine, The First Affiliated Hospital, China Medical University, Shenyang, China

Funder

The Mega-projects of National Science Research for the 13th Five-Year Plan

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/JVI.01655-19

Reference8 articles.

1. SAMHD1 restricts HIV-1 infection in resting CD4+ T cells

2. Kinetics of Human Immunodeficiency Virus Type 1 Decay following Entry into Resting CD4 + T Cells

3. Highly purified quiescent human peripheral blood CD4+ T cells are infectible by human immunodeficiency virus but do not release virus after activation

4. Membrane metalloprotease TRABD2A restricts HIV-1 progeny production in resting CD4+ T cells by degrading viral Gag polyprotein

5. Heavy metal protease takes a tiki torch to HIV assembly

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