Nectin-1 Is an Entry Mediator for Varicella-Zoster Virus Infection of Human Neurons-Reference-Cited by-同舟云学术

Nectin-1 Is an Entry Mediator for Varicella-Zoster Virus Infection of Human Neurons

Published:2021-10-27 Issue:22 Volume:95 Page:
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:J Virol

Author:

Rajbhandari Labchan¹,Shukla Priya¹,Jagdish Balaji¹,Mandalla Abby¹,Li Qingxue²,Ali Mir A.²,Lee Hojae³,Lee Gabsang³⁴,Sadaoka Tomohiko⁵^ORCID,Cohen Jeffrey I.²,Venkatesan Arun¹^ORCID

Affiliation:

1. Division of Neuroimmunology and Neurological Infections, Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

2. Medical Virology Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA

3. Institute for Cell Engineering, Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

4. The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

5. Division of Clinical Virology, Center for Infectious Diseases, Kobe University Graduate School of Medicine, Kobe, Japan

Abstract

Varicella-zoster virus (VZV) causes chickenpox, gains access to neurons during primary infection where it resides lifelong, and can later be reactivated. Reactivation is associated with shingles and postherpetic neuralgia, as well as with severe neurologic complications, including vasculitis and encephalitis.

Funder

HHS | NIH | National Institute of Neurological Disorders and Stroke

Tokyo Biochemical Research Foundation

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/JVI.01227-21

Reference53 articles.

1. Molecular mechanisms of varicella zoster virus pathogenesis

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