Hematopoietic Precursor Cells Transiently Reestablish Permissiveness for XInactivation
Author:
Affiliation:
1. Research Institute of Molecular Pathology, Vienna Biocenter, Dr. Bohr-Gasse 7, 1030 Vienna, Austria
2. Whitehead Institute for Biomedical Research, Nine Cambridge Center, Cambridge Massachusetts 02142
Abstract
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.00810-06
Reference50 articles.
1. Bacher C. P. M. Guggiari B. Brors S. Augui P. Clerc P. Avner R. Eils and E. Heard. 2006. Transient colocalization of X-inactivation centres accompanies the initiation of X inactivation. Nat. Cell. Biol.
2. Balciunaite, G., R. Ceredig, S. Massa, and A. G. Rolink. 2005 . A B220+ CD117+ CD19− hematopoietic progenitor with potent lymphoid andmyeloid developmental potential. Eur. J. Immunol.2019-2030.
3. Bassing, C. H., W. Swat, and F. W. Alt. 2002 . The mechanism and regulation of chromosomal V(D)J recombination. Cell109(Suppl.):S45-S55.
4. Borsani, G., R. Tonlorenzi, M. C. Simmler, L. Dandolo, D. Arnaud, V. Capra, M. Grompe, A. Pizzuti, D. Muzny, C. Lawrence, et al. 1991 . Characterization of a murine gene expressed from the inactive X chromosome. Nature351:325-329.
5. Brockdorff, N. 2002. X-chromosome inactivation: closing in on proteins that bind Xist RNA. Trends Genet.18:352-358.
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