IRF-3 Activation by Sendai Virus Infection Is Required for Cellular Apoptosis and Avoidance of Persistence
Author:
Affiliation:
1. Department of Molecular Genetics, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio 44195
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.00954-08
Reference3 articles.
1. Nishio, M., A. Nagata, M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2004. Recombinant Sendai viruses with L1618V mutation in their L polymerase protein establish persistent infection, but not temperature sensitivity. Virology329:289-301.
2. Nishio, M., M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2003. Characterization of Sendai virus persistently infected L929 cells and Sendai virus pi strain: recombinant Sendai viruses having Mpi protein shows lower cytotoxicity and are incapable of establishing persistent infection. Virology314:110-124.
3. Nishio, M., A. Nagata, A. Yamamoto, M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2006. The properties of recombinant Sendai virus having the P gene of Sendai virus pi strain derived from BHK cells persistently infected with Sendai virus. Med. Microbiol. Immunol.195:151-158.
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