Modulation and Utilization of Host Cell Phosphoinositides by Salmonella spp
Author:
Affiliation:
1. Laboratory of Intracellular Parasites, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, Hamilton, Montana 59840
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.72.8.4331-4335.2004
Reference59 articles.
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3. Brumell, J. H., and S. Grinstein. 2003. Role of lipid-mediated signal transduction in bacterial internalization. Cell. Microbiol.5:287-297.
4. Disruption of the Salmonella-Containing Vacuole Leads to Increased Replication of Salmonella enterica Serovar Typhimurium in the Cytosol of Epithelial Cells
5. Carricaburu, V., K. A. Lamia, E. Lo, L. Favereaux, B. Payrastre, L. C. Cantley, and L. E. Rameh. 2003. The phosphatidylinositol (PI)-5-phosphate 4-kinase type II enzyme controls insulin signaling by regulating PI-3,4,5-trisphosphate degradation. Proc. Natl. Acad. Sci. USA100:9867-9872.
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