TRIM22 Inhibits HIV-1 Transcription Independently of Its E3 Ubiquitin Ligase Activity, Tat, and NF- B-Responsive Long Terminal Repeat Elements-Reference-Cited by-同舟云学术

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TRIM22 Inhibits HIV-1 Transcription Independently of Its E3 Ubiquitin Ligase Activity, Tat, and NF- B-Responsive Long Terminal Repeat Elements

Published:2011-02-23 Issue:10 Volume:85 Page:5183-5196
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:Journal of Virology

Author:

Kajaste-Rudnitski A.,Marelli S. S.,Pultrone C.,Pertel T.,Uchil P. D.,Mechti N.,Mothes W.,Poli G.,Luban J.,Vicenzi E.

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Reference81 articles.

1. HIV-1 Infection of promonocytic U937 and U1 cell lines: clonal distribution of innate restriction factors;Alfano,2007

2. Susceptibility to infection by the human immunodeficiency virus (HIV) correlates with T4 expression in a parental monocytoid cell line and its subclones;Asjo;Virology,1987

3. The interferon response inhibits HIV particle production by induction of TRIM22;Barr;PLoS Pathog.,2008

4. Crystal structure of NFAT bound to the HIV-1 LTR tandem κB enhancer element;Bates;Structure,2008

5. Regulatory functions of ubiquitination in the immune system;Ben-Neriah;Nat. Immunol.,2002

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