Granulocytic Ehrlichiosis in Mice Deficient in Phagocyte Oxidase or Inducible Nitric Oxide Synthase-Reference-Cited by-同舟云学术

Granulocytic Ehrlichiosis in Mice Deficient in Phagocyte Oxidase or Inducible Nitric Oxide Synthase

Published:2000-07 Issue:7 Volume:68 Page:4361-4362
ISSN:0019-9567
Container-title:Infection and Immunity
language:en
Short-container-title:Infect Immun

Author:

Banerjee Rila¹,Anguita Juan¹,Fikrig Erol¹

Affiliation:

1. Section of Rheumatology, Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut 06520

Abstract

ABSTRACT Mice deficient in phox (gp91 phox−/− ) or NOS2 (NOS2 −/− ) were infected with the agent of human granulocytic ehrlichiosis (HGE) to evaluate the importance of these pathways in the eradication of HGE bacteria. NOS2 −/− mice had delayed clearance of the HGE agent in comparison to control or gp91 phox−/− mice, suggesting that reactive nitrogen intermediates play a role in the early control of HGE.

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Immunology,Microbiology,Parasitology

Link

https://journals.asm.org/doi/pdf/10.1128/IAI.68.7.4361-4362.2000

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