DeSUMOylation of Apoptosis Inhibitor 5 by Avibirnavirus VP3 Supports Virus Replication-Reference-Cited by-同舟云学术

DeSUMOylation of Apoptosis Inhibitor 5 by Avibirnavirus VP3 Supports Virus Replication

Published:2021-08-31 Issue:4 Volume:12 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Deng Tingjuan¹,Hu Boli¹,Wang Xingbo¹,Yan Yan¹,Zhou Jianwei¹,Lin Lulu¹,Xu Yuting¹,Zheng Xiaojuan¹,Zhou Jiyong¹²^ORCID

Affiliation:

1. MOA Key Laboratory of Animal Virology, Zhejiang University Center for Veterinary Sciences, Hangzhou, Zhejiang, People’s Republic of China

2. State Key Laboratory for Diagnosis and Treatment of Infectious Diseases, First Affiliated Hospital, Zhejiang University, Hangzhou, Zhejiang, People’s Republic of China

Abstract

Apoptosis inhibitor 5 (API5) is a nuclear protein initially identified for its antiapoptotic function. However, so far, posttranslational modification of API5 is unclear.

Funder

National Natural Science Foundation of China

China Agricultural Research System

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mBio.01985-21

Reference79 articles.

1. SUMO2/3 modification of activating transcription factor 5 (ATF5) controls its dynamic translocation at the centrosome

2. SUMOylation inhibits FOXM1 activity and delays mitotic transition