Caspase-Mediated Regulation and Cellular Heterogeneity of the cGAS/STING Pathway in Kaposi’s Sarcoma-Associated Herpesvirus Infection-Reference-Cited by-同舟云学术

Caspase-Mediated Regulation and Cellular Heterogeneity of the cGAS/STING Pathway in Kaposi’s Sarcoma-Associated Herpesvirus Infection

Published:2022-12-20 Issue:6 Volume:13 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Tabtieng Tate¹²,Lent Rachel C.¹³,Kaku Machika¹⁴,Monago Sanchez Alvaro¹⁵,Gaglia Marta Maria¹²³⁴⁶^ORCID

Affiliation:

1. Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, USA

2. Program in Biochemistry, Tufts University Graduate School of Biomedical Sciences, Boston, Massachusetts, USA

3. Program in Molecular Microbiology, Tufts University Graduate School of Biomedical Sciences, Boston, Massachusetts, USA

4. Program in Immunology, Tufts University Graduate School of Biomedical Sciences, Boston, Massachusetts, USA

5. Faculty of Experimental Sciences, Universidad Francisco de Vitoria, Madrid, Spain

6. Institute for Molecular Virology and Department of Medical Microbiology and Immunology, University of Wisconsin—Madison, Wisconsin, USA

Abstract

Type I interferons are key factors that dictate the outcome of infectious and inflammatory diseases. Thus, intricate cellular regulatory mechanisms are in place to control IFN responses.

Funder

American Cancer Society

HHS | NIH | National Institute of General Medical Sciences

Tufts Graduate School of Biomedical Sciences

HHS | NIH | National Cancer Institute

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mbio.02446-22

Reference52 articles.

1. Innate Immune Sensing and Signaling of Cytosolic Nucleic Acids

2. Interferon-stimulated genes: roles in viral pathogenesis

3. Ten Strategies of Interferon Evasion by Viruses

4. Caspase-Dependent Suppression of Type I Interferon Signaling Promotes Kaposi's Sarcoma-Associated Herpesvirus Lytic Replication

5. Cytoplasmic isoforms of Kaposi sarcoma herpesvirus LANA recruit and antagonize the innate immune DNA sensor cGAS

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