Gallid Herpesvirus 1 Initiates Apoptosis in Uninfected Cells through Paracrine Repression of p53
Author:
Affiliation:
1. State Key Laboratory of Veterinary Biotechnology, Harbin Veterinary Research Institute, the Chinese Academy of Agricultural Sciences, Harbin, People's Republic of China
Abstract
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.00529-18
Reference59 articles.
1. Persistence and Expression of the Herpes Simplex Virus Genome in the Absence of Immediate-Early Proteins
2. ICP0 Gene Expression Is a Herpes Simplex Virus Type 1 Apoptotic Trigger
3. Direct activation of RIP3/MLKL-dependent necrosis by herpes simplex virus 1 (HSV-1) protein ICP6 triggers host antiviral defense
4. Apoptosis During Herpes Simplex Virus Infection
5. Modulation of sFas indicates apoptosis in human herpes simplex encephalitis
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2. Integrative RNA-seq and ChIP-seq analysis unveils metabolic regulation as a conserved antiviral mechanism of chicken p53;Microbiology Spectrum;2024-08-06
3. Characterization of the Effects of Host p53 and Fos on Gallid Alpha Herpesvirus 1 Replication;Genes;2023-08-12
4. P53 maintains gallid alpha herpesvirus 1 replication by direct regulation of nucleotide metabolism and ATP synthesis through its target genes;Frontiers in Microbiology;2022-11-24
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