Suppression of Extracellular Signal-Regulated Kinase Activity in Herpes Simplex Virus 1-Infected Cells by the Us3 Protein Kinase

Author:

Chuluunbaatar U.,Roller R.,Mohr I.

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Reference58 articles.

1. Herpes simplex virus type 1 ICP4 deletion mutant virus d120 infection failed to induce apoptosis in nerve growth factor-differentiated PC12 cells;Aiamkitsumrit;J. Neurovirol.,2007

2. Herpes simplex virus protein kinase US3 activates and functionally overlaps protein kinase A to block apoptosis;Benetti;Proc. Natl. Acad. Sci. U. S. A.,2004

3. The activation of p38 and apoptosis by the inhibition of ERK is antagonized by the phosphoinositide 3-kinase/Akt pathway;Berra;J. Biol. Chem.,1998

4. Inhibition of mTORC1 leads to MAPK path-way activation through a PI3K-dependent feedback loop in human cancer;Carracedo;J. Clin. Invest.,2008

5. The PTEN-PI3K pathway: of feedbacks and cross-talks;Carracedo;Oncogene,2008

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