IL-1R1-Dependent Signals Improve Control of Cytosolic Virulent Mycobacteria In Vivo-Reference-Cited by-同舟云学术

IL-1R1-Dependent Signals Improve Control of Cytosolic Virulent Mycobacteria In Vivo

Published:2021-05-05 Issue: Volume: Page:
ISSN:2379-5042
Container-title:mSphere
language:en
Short-container-title:mSphere

Author:

van der Niet Sanne¹,van Zon Maaike²,de Punder Karin²,Grootemaat Anita¹,Rutten Sofie¹,Moorlag Simone J. C. F. M.²,Houben Diane²,van der Sar Astrid M.³,Bitter Wilbert³,Brosch Roland⁴^ORCID,Hernandez Pando Rogelio⁵,Pena Maria T.⁶,Peters Peter J.²,Reits Eric A.¹,Mayer-Barber Katrin D.⁷,van der Wel Nicole N.¹²^ORCID

Affiliation:

1. Electron Microscopy Centre Amsterdam, Amsterdam University Medical Centre AMC, Amsterdam, The Netherlands

2. Netherlands Cancer Institute, Amsterdam, The Netherlands

3. Amsterdam University Medical Centre VUMC, Amsterdam, The Netherlands

4. Unit for Integrated Mycobacterial Pathogenomics, CNRS UMR 3525, Paris, France

5. National Institute of Medical Sciences and Nutrition, Mexico City, Mexico

6. Department of Health and Human Services, Health Resources and Services Administration, Healthcare Systems Bureau, National Hansen’s Disease Programs, Baton Rouge, Louisiana, USA

7. Inflammation and Innate Immunity Unit, Laboratory of Clinical Immunology and Microbiology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA

Abstract

For decades, Mycobacterium tuberculosis has been one of the deadliest pathogens known. Despite infecting approximately one-third of the human population, no effective treatment or vaccine is available.

Funder

HHS | National Institutes of Health

Institut Pasteur

Publisher

American Society for Microbiology

Subject

Molecular Biology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mSphere.00153-21

Reference67 articles.

1. World Health Organization. 2019. Global tuberculosis report 2019: fact sheet. World Health Organization, Geneva, Switzerland. https://www.who.int/teams/global-tuberculosis-programme/tb-reports/global-report-2019.

2. Dissecting How CD4 T Cells Are Lost During HIV Infection

3. The Macrophage: A Disputed Fortress in the Battle against Mycobacterium tuberculosis

4. The innate immune response in human tuberculosis

5. Growth of Mycobacterium tuberculosis in vivo segregates with host macrophage metabolism and ontogeny

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