Bcl-3 Expression Promotes Cell Survival following Interleukin-4 Deprivation and Is Controlled by AP1 and AP1-Like Transcription Factors
Author:
Affiliation:
1. Centro Nacional de Biotecnologı́a, Department of Immunology and Oncology, UAM, E-28049 Madrid, Spain,1 and
2. Ludwig Institute for Cancer Research, UCL 74.59, B-1200, Brussels, Belgium2
Abstract
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.20.10.3407-3416.2000
Reference47 articles.
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2. NF-κB: ten years after;Baeuerle P. A.;Cell,1996
3. The NF-κB and Iκ-B proteins: new discoveries and insights;Baldwin A. S.;Annu. Rev. Immunol.,1996
4. Iκ-B interacts with the nuclear localization sequences of the subunits of NF-κB: a mechanism of cytoplasmic retention;Beg A. A.;Genes Dev.,1992
5. Mouse Bcl-3: cDNA structure, mapping and stage-dependent expression in B lymphocytes;Bhatia K.;Oncogene,1991
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