Hepatitis C Virus-Induced Exosomal MicroRNAs and Toll-Like Receptor 7 Polymorphism Regulate B-Cell Activating Factor

Author:

Liao Tsai-Ling123ORCID,Chen Yi-Ming1234,Hsieh Shie-Liang56,Tang Kuo-Tung4,Chen Der-Yuan7,Yang Ying-Ying8,Liu Hung-Jen23910ORCID,Yang Sheng-Shun3111213

Affiliation:

1. Department of Medical Research, Taichung Veterans General Hospital, Taichung, Taiwan

2. Rong Hsing Research Center for Translational Medicine, National Chung Hsing University, Taichung, Taiwan

3. Ph.D. Program in Translational Medicine, National Chung Hsing University, Taichung, Taiwan

4. Division of Allergy, Immunology, and Rheumatology, Taichung Veterans General Hospital, Taichung, Taiwan

5. Genomics Research Center, Academia Sinica, Taipei, Taiwan

6. Faculty of Medicine, National Yang Ming University, Taipei, Taiwan

7. Translational Medicine Laboratory, Rheumatology and Immunology Center, China Medical University Hospital, Taichung, Taiwan

8. Division of Gastroenterology and Hepatology, Taipei Veterans General Hospital, Taipei, Taiwan

9. Institute of Molecular Biology, National Chung Hsing University, Taichung, Taiwan

10. The iEGG and Animal Biotechnology Center, National Chung Hsing University, Taichung, Taiwan

11. Division of Gastroenterology and Hepatology, Department of Internal Medicine, Taichung Veterans General Hospital, Taichung, Taiwan

12. School of Medicine, Chung Shan Medical University, Taichung, Taiwan

13. Institute of Biomedical Sciences, National Chung Hsing University, Taichung, Taiwan

Abstract

HCV remains an important cause of liver disease worldwide. Accumulating evidence has demonstrated that HCV infection is associated with B cell lymphoproliferative disorders such as MC.

Funder

Taichung Veterans General Hospital

Academia Sinic

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

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