Adaptation in Gitelman Syndrome: “We Just Want to Pump You Up”
Author:
Publisher
American Society of Nephrology (ASN)
Subject
Transplantation,Nephrology,Critical Care and Intensive Care Medicine,Epidemiology
Reference25 articles.
1. Rare independent mutations in renal salt handling genes contribute to blood pressure variation
2. Localization of Tubular Adaptation to Renal Sodium Loss in Gitelman Syndrome
3. A new familial disorder characterized by hypokalemia and hypomagnesemia;Gitelman;Trans Am Assoc Phys,1966
4. Use of calcium excretion values to distinguish two forms of primary renal tubular hypokalemic alkalosis: Bartter and Gitelman syndromes
5. Gitelman's variant of Barter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na–Cl cotransporter
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1. Gitelman syndrome: an analysis of the underlying pathophysiologic mechanisms of acid–base and electrolyte abnormalities;International Urology and Nephrology;2017-07-25
2. Pathophysiology and clinical presentations of salt-losing tubulopathies;Pediatric Nephrology;2015-07-16
3. Distal Convoluted Tubule;Comprehensive Physiology;2014-12-23
4. The sodium chloride cotransporter SLC12A3: new roles in sodium, potassium, and blood pressure regulation;Pflügers Archiv - European Journal of Physiology;2013-12-06
5. Phosphorylation Regulates NCC Stability and Transporter Activity In Vivo;Journal of the American Society of Nephrology;2013-07-05
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