Right–side predominance of the central amygdala activation in trigeminal inflammatory pain model

Author:

Takahashi Yukari1,Miyazawa Yuta1,Arimura Daigo1,Sugimura Yae K1,Kato Fusao1

Affiliation:

1. Center for Neuroscience of Pain, Jikei University School of Medicine

Publisher

Japanese Association for the Study of Pain

Subject

General Medicine

Reference26 articles.

1. 1) Arimura, D., Shinohara, K., Takahashi, Y., Arimura, D., Shinohara, K., Takahashi, Y., Sugimura, Y.K., Sugimoto, M., Tsurugizawa, T., Marumo, K., Kato, F., Primary Role of the Amygdala in Spontaneous Inflammatory Pain– Associated Activation of Pain Networks – A Chemogenetic Manganese–Enhanced MRI Approach, Front. Neural Circuits, 13 (2019) 58.

2. 2) Bernard, J.F., Alden, M., Besson, J.M., The organization of the efferent projections from the pontine parabrachial area to the amygdaloid complex: a Phaseolus vulgaris leucoagglutinin (PHA–L) study in the rat, J. Comp. Neurol., 329 (1993) 201–229.

3. 3) Bernard, J.F., Huang, G.F., Besson, J.M., Effect of noxious somesthetic stimulation on the activity of neurons of the nucleus centralis of the amygda­la, Brain Res., 523 (1990) 347–350.

4. 4) Bernard, J.F., Peschanski, M., Besson, J.M., A possible spino (trigemino)–ponto–amygdaloid pathway for pain, Neurosci. Lett., 100 (1989) 83–88.

5. 5) Burgmer, M., Gaubitz, M., Konrad, C., Wrenger, M., Hilgart, S., Heuft, G., Pfleiderer, B., Decreased gray matter volumes in the cingulo–frontal cortex and the amygdala in patients with fibromyalgia, Psychosom. Med., 71 (2009) 566–573.

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