Cancer genesis: stem tumour cells as an MHC-null/HSP70 – very high ‘primordial self’ escaping both MHC-restricted and MHC-non- restricted immunesurveillance-Reference-Cited by-同舟云学术

Cancer genesis: stem tumour cells as an MHC-null/HSP70 – very high ‘primordial self’ escaping both MHC-restricted and MHC-non- restricted immunesurveillance

Published:2001-06 Issue:6 Volume:56 Page:724-730
ISSN:0306-9877
Container-title:Medical Hypotheses
language:en
Short-container-title:Medical Hypotheses

Author:

Manzo G.

Publisher

Elsevier BV

Subject

General Medicine

Reference39 articles.

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3. HLA class I gene expression on human primary tumors and autologous metastases: demonstration of selective losses of HLA antigens on colorectal, gastric and laryngeal carcinomas;Lopez-Nevot;Br J Cancer,1989

4. Immunological correlations between ontogenesis and oncogenesis: theoretical implications and suggestions for tumor immunotherapy;Manzo;Med Hypotheses,1992

5. Altered protein synthesis in p53 null and hemizygous transgenic mouse embryonic fibroblasts;He;Appl Theor Electophor,1995

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2. Glioma Stem Cell Research for the Development of Immunotherapy;Advances in Experimental Medicine and Biology;2012

3. Unexpected deeds of familiar proteins: Interplay of Hsp70, PGRP S / Tag7, and S100A4 / Mts1 in host vs. cancer combat;Cell Cycle;2010-02-15

4. Antigen-Specific T-Cell Response from Dendritic Cell Vaccination Using Cancer Stem-Like Cell-Associated Antigens;Stem Cells;2009-04-23