p63 null mutation protects mouse oocytes from radio-induced apoptosis-Reference-Cited by-同舟云学术

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p63 null mutation protects mouse oocytes from radio-induced apoptosis

Published:2007-12-25 Issue:1 Volume:135 Page:3-12
ISSN:1470-1626
Container-title:Reproduction
language:en
Short-container-title:Reproduction

Author:

Livera G.,Petre-Lazar B.,Guerquin M.-J.,Trautmann E.,Coffigny H.,Habert R.

Publisher

Bioscientifica

Subject

Cell Biology,Obstetrics and Gynecology,Endocrinology,Embryology,Reproductive Medicine

Reference20 articles.

1. The role of the tumor suppressor p53 in spermatogenesis

2. p73 and p63 Are Homotetramers Capable of Weak Heterotypic Interactions with Each Other but Not with p53

3. A Subset of Tumor-Derived Mutant Forms of p53 Down-Regulate p63 and p73 through a Direct Interaction with the p53 Core Domain

4. TAp63α induces apoptosis by activating signaling via death receptors and mitochondria

5. Unaltered Development of the Initial Follicular Waves and Normal Pubertal Onset in Female Rats after Neonatal Deletion of the Follicular Reserve

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1. The role of declining ataxia-telangiectasia-mutated (ATM) function in oocyte aging;Cell Death Discovery;2024-06-25

2. TP63 truncating mutation causes increased cell apoptosis and premature ovarian insufficiency by enhanced transcriptional activation of CLCA2;Journal of Ovarian Research;2024-03-25

3. p53 regulates diverse tissue-specific outcomes to endogenous DNA damage in mice;Nature Communications;2024-03-21

4. Cathepsin B regulates ovarian reserve quality and quantity via mitophagy by modulating IGF1R turnover;2024-02-15

5. Single-cell and bulk transcriptional profiling of mouse ovaries reveals novel genes and pathways associated with DNA damage response in oocytes;2024-02-05

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