Molecular and Genetic Aspects of Lung Cancer
Author:
Publisher
Informa UK Limited
Subject
Biotechnology
Link
http://www.tandfonline.com/doi/pdf/10.5504/BBEQ.2013.0062
Reference102 articles.
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3. Inactivation of the p16 gene by hypermethylation and loss of heterozygosity in adenocarcinoma of the lung
4. Fhit, a Putative Tumor Suppressor in Humans, Is a Dinucleoside 5‘,5‘ ‘‘-P1,P3-Triphosphate Hydrolase
5. Aberrant methylation of p16INK4a is an early event in lung cancer and a potential biomarker for early diagnosis
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