Similar voltage-sensor movement in spHCN channels can cause closing, opening, or inactivation
Author:
Affiliation:
1. Department of Physiology and Biophysics, University of Miami 1 , Miami, FL, USA
2. Miller School of Medicine 1 , Miami, FL, USA
Abstract
Funder
National Institutes of Health
Publisher
Rockefeller University Press
Subject
Physiology
Link
https://rupress.org/jgp/article-pdf/doi/10.1085/jgp.202213170/1447533/jgp_202213170.pdf
Reference33 articles.
1. Integrated allosteric model of voltage gating of HCN channels;Altomare;J. Gen. Physiol.,2001
2. Hyperpolarization-activated cation channels: From genes to function;Biel;Physiol. Rev.,2009
3. How does adrenaline accelerate the heart?;Brown;Nature,1979
4. Slow conformational changes of the voltage sensor during the mode shift in hyperpolarization-activated cyclic-nucleotide-gated channels;Bruening-Wright;J. Neurosci.,2007
5. Properties of hyperpolarization-activated pacemaker current defined by coassembly of HCN1 and HCN2 subunits and basal modulation by cyclic nucleotide;Chen;J. Gen. Physiol.,2001
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1. Different fluorescent labels report distinct components of spHCN channel voltage sensor movement;2024-01-26
2. Direct regulation of the voltage sensor of HCN channels by membrane lipid compartmentalization;Nature Communications;2023-10-18
3. Direct Regulation of the Voltage Sensor of HCN Channels by Membrane Lipid Compartmentalization;2023-06-15
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