Di-n-Butyl Phthalate Induces Multinucleated Germ Cells in the Rat Fetal Testis Through a Nonproliferative Mechanism1
Author:
Publisher
Oxford University Press (OUP)
Subject
Cell Biology,General Medicine,Reproductive Medicine
Link
http://academic.oup.com/biolreprod/article-pdf/93/5/110
Reference63 articles.
1. Dose-response assessment of fetal testosterone production and gene expression levels in rat testes following in utero exposure to diethylhexyl phthalate, diisobutyl phthalate, diisoheptyl phthalate, and diisononyl phthalate;Toxicol Sci,2011
2. Perinatal exposure to the phthalates DEHP, BBP, and DINP, but not DEP, DMP, or DOTP, alters sexual differentiation of the male rat;Toxicol Sci,2000
3. Fetal testosterone insufficiency and abnormal proliferation of Leydig cells and gonocytes in rats exposed to di(n-butyl) phthalate;Reprod Toxicol,2002
4. Fetal mouse phthalate exposure shows that Gonocyte multinucleation is not associated with decreased testicular testosterone;Toxicol Sci,2007
5. The plasticizer diethylhexyl phthalate induces malformations by decreasing fetal testosterone synthesis during sexual differentiation in the male rat;Toxicol Sci,2000
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4. The role of small GTPases in bisphenol AF-induced multinucleation in comparison with dibutyl phthalate in the male germ cells;Toxicological Sciences;2023-01-10
5. Interaction between mono-(2-ethylhexyl) phthalate and retinoic acid alters Sertoli cell development during fetal mouse testis cord morphogenesis;Current Research in Toxicology;2022
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