SMAD3 Activation: A Converging Point of Dysregulated TGF-Beta Superfamily Signaling and Genetic Aberrations in Granulosa Cell Tumor Development?-Reference-Cited by-同舟云学术

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SMAD3 Activation: A Converging Point of Dysregulated TGF-Beta Superfamily Signaling and Genetic Aberrations in Granulosa Cell Tumor Development?

Published:2016-09-28 Issue:5 Volume:95 Page:105-105
ISSN:0006-3363
Container-title:Biology of Reproduction
language:en
Short-container-title:Biology of Reproduction

Author:

Fang X.,Gao Y.,Li Q.

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,General Medicine,Reproductive Medicine

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A novel TGFbeta/TGILR axis mediates crosstalk between cancer-associated fibroblasts and tumor cells to drive gastric cancer progression;Cell Death & Disease;2024-05-28

2. The Role of Smad3 in the Realization of the Growth Potential of Different Types of Neural Progenitor Cells and the Secretory Function of Neuroglia;Bulletin of Experimental Biology and Medicine;2024-05

3. Sertoli Cell-Specific Activation of Transforming Growth Factor Beta Receptor 1 Leads to Testicular Granulosa Cell Tumor Formation;Cells;2023-11-27

4. Oocyte‐secreted factor TGFB2 enables mouse cumulus cell expansion in vitro;Molecular Reproduction and Development;2022-09-21

5. Physosmotic Induction of Chondrogenic Maturation Is TGF-β Dependent and Enhanced by Calcineurin Inhibitor FK506;International Journal of Molecular Sciences;2022-05-04

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