Decreased Langerhans Cell Responses to IL-36γ: Altered Innate Immunity in Patients with Recurrent Respiratory Papillomatosis-Reference-Cited by-同舟云学术

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Decreased Langerhans Cell Responses to IL-36γ: Altered Innate Immunity in Patients with Recurrent Respiratory Papillomatosis

Published:2014-01 Issue:1 Volume:20 Page:372-380
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

DeVoti James,Hatam Lynda,Lucs Alexandra,Afzal Ali,Abramson Allan,Steinberg Bettie,Bonagura Vincent

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.2119/molmed.2014.00098.pdf

Reference66 articles.

1. DeVoti JA, et al. (2004) Failure of gamma interferon but not interleukin-10 expression in response to human papillomavirus type 11 E6 protein in respiratory papillomatosis. Clin. Diagn. Lab. Immunol. 11:538–47.

2. Bonagura VR, et al. (2009) KIR3DS1, KIR2DS1, and KIR2DS5 protect against the development of severe recurrent respiratory papillomatosis (RRP) in HPV-6/11-Infected patients. J. Allergy Clin. Immunol. 123:S165.

3. Bonagura VR, et al. (2010) Recurrent respiratory papillomatosis: a complex defect in immune responsiveness to human papillomavirus-6 and -11. APMIS. 118:455–70.

4. DeVoti JA, et al. (2008) Immune dysregulation and tumor-associated gene changes in recurrent respiratory papillomatosis: A paired microarray analysis. Mol. Med. 14:608–17.

5. Vigne S, et al. (2012) IL-36 signaling amplifies Th1 responses by enhancing proliferation and Th1 polarization of naive CD4+ T cells. Blood. 120:3478–87.

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