Suppression of parotid acinar cell dysfunction by the free radical scavenger 3-methyl-1-phenyl-2-pyrazolin-5-one-Reference-Cited by-同舟云学术

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Suppression of parotid acinar cell dysfunction by the free radical scavenger 3-methyl-1-phenyl-2-pyrazolin-5-one

Published:2019 Issue:3 Volume:61 Page:475-480
ISSN:1343-4934
Container-title:Journal of Oral Science
language:en
Short-container-title:J Oral Sci

Author:

Sakurai Hajime¹,Yokoyama Megumi²³,Katsumata-Kato Osamu²³,Fujita-Yoshigaki Junko²³

Affiliation:

1. Division of Cellular Physiology, Nihon University Graduate School of Dentistry at Matsudo

2. Department of Physiology, Nihon University School of Dentistry at Matsudo

3. Research Institute of Oral Science, Nihon University School of Dentistry at Matsudo

Publisher

Nihon University School of Dentistry

Subject

General Dentistry

Link

https://www.jstage.jst.go.jp/article/josnusd/61/3/61_18-0405/_pdf

Reference21 articles.

1. 1. Baum BJ (2000) Prospects for re-engineering salivary glands. Adv Dent Res 14, 84-88.

2. 2. Fujita-Yoshigaki J, Tagashira A, Yoshigaki T, Furuyama S, Sugiya H (2005) A primary culture of parotid acinar cells retaining capacity for agonists-induced amylase secretion and generation of new secretory granules. Cell Tissue Res 320, 455-464.

3. 3. Qi B, Fujita-Yoshigaki J, Michikawa H, Satoh K, Katsumata O, Sugiya H (2007) Differences in claudin synthesis in primary cultures of acinar cells from rat salivary gland are correlated with the specific three-dimensional organization of the cells. Cell Tissue Res 329, 59-70.

4. 4. Fujita-Yoshigaki J, Matsuki-Fukushima M, Sugiya H (2008) Inhibition of Src and p38 MAP kinases suppresses the change of claudin expression induced on dedifferentiation of primary cultured parotid acinar cells. Am J Physiol Cell Physiol 294, C774-785.

5. 5. Moriyama S, Yokoyama M, Katsumata-Kato O (2015) Enhancement of Src family kinase activity is essential for p38 MAP kinase-mediated dedifferentiation signal of parotid acinar cells. Int J Oral-Med Sci 14, 33-40.

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