Ad libitum feeding triggers puberty onset associated with increases in arcuate Kiss1 and Pdyn expression in growth-retarded rats
Author:
Affiliation:
1. Laboratory of Animal Reproduction, Graduate School of Bioagricultural Sciences, Nagoya University, Nagoya 464-8601, Japan
Publisher
Japanese Society of Animal Reproduction
Subject
Animal Science and Zoology
Link
https://www.jstage.jst.go.jp/article/jrd/65/5/65_2019-048/_pdf
Reference63 articles.
1. 1. Sisk CL, Foster DL. The neural basis of puberty and adolescence. Nat Neurosci 2004; 7: 1040–1047.
2. 2. Uenoyama Y, Inoue N, Ieda N, Pheng V, Maeda K-I, Tsukamura H. Maturation and physiology of hypothalamic regulation of the gonadal axis. In: Kumanov P, Agarwal A (eds.), Puberty: Physiology and Abnormalities. Springer 2016: 1–11.
3. 3. Uenoyama Y, Pheng V, Tsukamura H, Maeda KI. The roles of kisspeptin revisited: inside and outside the hypothalamus. J Reprod Dev 2016; 62: 537–545.
4. 4. Day ML, Imakawa K, Zalesky DD, Kittok RJ, Kinder JE. Effects of restriction of dietary energy intake during the prepubertal period on secretion of luteinizing hormone and responsiveness of the pituitary to luteinizing hormone-releasing hormone in heifers. J Anim Sci 1986; 62: 1641–1648.
5. 5. Foster DL, Olster DH. Effect of restricted nutrition on puberty in the lamb: patterns of tonic luteinizing hormone (LH) secretion and competency of the LH surge system. Endocrinology 1985; 116: 375–381.
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3. Conditional Oprk1-dependent Kiss1 deletion in kisspeptin neurons caused estrogen-dependent LH pulse disruption and LH surge attenuation in female rats;Scientific Reports;2023-11-22
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