Impaired Postnatal Development in C/EBP.BETA.-deficient Mice-Reference-Cited by-同舟云学术

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Impaired Postnatal Development in C/EBP.BETA.-deficient Mice

Published:2006 Issue:5 Volume:52 Page:645-649
ISSN:0916-8818
Container-title:Journal of Reproduction and Development
language:en
Short-container-title:Journal of Reproduction and Development

Author:

BAI Tao¹²,TANAKA Tetsuji¹,YUKAWA Kazunori³,UMESAKI Naohiko¹,MATSUMOTO Makoto⁴,AKIRA Shizuo⁴

Affiliation:

1. Department of Obstetrics and Gynecology, Wakayama Medical University

2. Department of Obstetrics and Gynecology, The Second Affiliated Hospital of China Medical University

3. Department of Physiology, Wakayama Medical University

4. Department of Host Defence, Research Institute of Microbial Disease, Osaka University

Publisher

Japanese Society of Animal Reproduction

Subject

Animal Science and Zoology

Link

http://www.jstage.jst.go.jp/article/jrd/52/5/52_5_645/_pdf

Reference6 articles.

1. Dimerization specificity of the leucine zipper-containing bZIP motif on DNA binding: prediction and rational design.

2. Lymphoproliferative disorder and imbalanced T-helper response in C/EBP beta-deficient mice.

3. Targeted disruption of the NF-IL6 gene discloses its essential role in bacteria killing and tumor cytotoxicity by macrophages

4. Defective adipocyte differentiation in mice lacking the C/EBPbeta and/or C/EBPdelta gene

5. An essential role for C/EBPβ in female reproduction

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Prospective signs of cleidocranial dysplasia in Cebpb deficiency;Journal of Biomedical Science;2014-05-13

2. Neither Absence Nor Excess of FGF23 Disturbs Murine Fetal-Placental Phosphorus Homeostasis or Prenatal Skeletal Development and Mineralization;Endocrinology;2014-05-01

3. Phenotypes of CCAAT/enhancer-binding protein beta deficiency: hyperdontia and elongated coronoid process;Oral Diseases;2012-07-31

4. CCAAT/Enhancer-binding Protein β Promotes Osteoblast Differentiation by Enhancing Runx2 Activity with ATF4;Molecular Biology of the Cell;2008-12

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