Detection of left ventricular dysfunction after acute myocardial infarction: comparison of clinical, echocardiographic, and neurohormonal methods.

Author:

Choy A M,Darbar D,Lang C C,Pringle T H,McNeill G P,Kennedy N S,Struthers A D

Publisher

BMJ

Subject

Cardiology and Cardiovascular Medicine

Reference45 articles.

1. Left ventricular and systolic volume as the major determinant of survival after recovery from myocardial infarction;White, H.D.; Norris, R.M.; Brown, M.A.; Brandt, P.W.T.; Whitlock, R.M.L.; Wild, C.J.;Circulation,1987

2. Influence of chronic captopril therapy on the infarcted left ventricle of the rat;Pfeffer, J.M.; Pfeffer, M.A.; Braunwald, E.;Circ Res,1985

3. Treatment of patients with symptomless left ventricular dysfunction after myocardial infarction;Sharpe, N.; Murphy, J.; Smith, H.; Hannan, S.;Lancet,1988

4. Effect of captopril on progressive ventricular dilatation after anterior myocardial infarction;Pfeffer, M.A.; Lamas, G.A.; Vaughan, D.E.; Parisi, A.F.; Braunwald, E.;N Engl JMed,1988

5. Early prevention of left ventricular dysfunction after myocardial infarction with angiotensin-converting-enzyme inhibition;Sharpe, N.; Smith, H.; Murphy, J.; Greaves, S.; Hart, H.; Gamble, G.;Lancet,1991

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