Glutathione reductase activity, riboflavin status, and disease activity in rheumatoid arthritis.

Author:

Mulherin D M,Thurnham D I,Situnayake R D

Publisher

BMJ

Subject

General Biochemistry, Genetics and Molecular Biology,Immunology,Immunology and Allergy,Rheumatology

Reference16 articles.

1. Nitric oxide and arthritis;Stefanovic-Racic, M.; Stadler, J.; Evans, C.H.;Arthritis Rheum,1993

2. Biochemistry;Voet, D.; Voet, J.G.,1995

3. Method for the detection of a biochemical riboflavin deficiency. Stimulation ofNADPH2-dependent glutathione reductase from human erythrocytes by FAD in vitro. Investigations on the vitamin B2 status in healthy people and geriatric patients;IntJI Vitam Res,1970

4. Changes in blood glutathione concentrations, and in erythrocyte glutathione reductase and glutathione S-transferase activity after running training and after participation in contests;Evelo, C.T.A.; Palmen, N.G.M.; Artur, Y.; Janssen, G.M.E.;EurJ Appl Physiol,1992

5. D-penicillamine-induced increase in intracellular glutathione correlating to clinical response in rheumatoid arthritis;Munthe, E.; Kass, E.; Jellum, E.,1981

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