Studies of non-disjunction in trisomies 2, 7, 15, and 22: does the parental origin of trisomy influence placental morphology?-Reference-Cited by-同舟云学术

Studies of non-disjunction in trisomies 2, 7, 15, and 22: does the parental origin of trisomy influence placental morphology?

Published:1998-11-01 Issue:11 Volume:35 Page:924-931
ISSN:1468-6244
Container-title:Journal of Medical Genetics
language:en
Short-container-title:Journal of Medical Genetics

Author:

Zaragoza M V,Millie E,Redline R W,Hassold T J

Publisher

BMJ

Subject

Genetics(clinical),Genetics

Reference33 articles.

1. Trisomy in man;Hassold, T.J.; Jacobs, P.A.;Annu Rev Genet,1984

2. Recombination and maternal age-dependent nondisjunction: molecular studies of trisomy 16;Hassold, T.J.; Merrill, M.; Adkins, K.; Freeman, S.; Sherman, S.;Am JHum Genet,1995

3. Trisomy 18: studies of the parent and cell division of origin and the effect of aberrant recombination on nondisjunction;Fisher, J.M.; Harvey, J.F.; Morton, N.E.; Jacobs, P.A.;Am Hum Genet,1995

4. Susceptible chiasmate configurations of chromosome 21 predispose to non-disjunction in both maternal meiosis I and meiosis II;Lamb, N.E.; Freeman, S.B.; Savage-Austin, A.;Nat Genet,1996

5. Recombination and nondisjunction in humans and flies;Koehler, K.E.; Hawley, R.S.; Sherman, S.; Hassold, T.;Hum Mol Genet,1996

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