1. Immunofluorescence studies in a caseofGoodpasture's sydrome;Sturgill, B.C.; Westervelt, F.B.;JAMA,1965
2. Immunologic studies concerning the pulmonary lesions in Gooda protein not involved in any immunological reaction, at the same sites as the immunoglobulins is evidence that this is the explanation in many of our cases (Table). The other mechanism is that an immunological reaction has occurred which is suggested by the presence of complement. However in cases in which C3 is present without Clq it is possible that complement could have been activated via the alternate pathway by aggregated IgA which may be pasture's syndrome;Koffler, D.; Sandson, J.; Carr, R.; Kunkel, H.G.;Am JPathol,1969
3. Variant "Goodpasture's syndrome"?. The need for immunologic criteria in rapidly progressive glomerulonephritis and hemorrhagic pneumonitis;Martinez, J.S.; Kohler, P.F.;Ann Intern Med,1971
4. Goodpasture's syndrome: two cases with contrasting early course and management;Teishman, S.; Briggs, W.A.; Knieser, M.R.; Enquist, R.W.;Am Rev Respir Dis,1976
5. IgA antibasement membrane nephritis with pulmonary hemorrhage;Border, W.A.; Baehler, R.W.; Bhathena, D.; Glassock, R.J.;Ann Intern Med,1979