Treatment of Bartter's syndrome in early childhood with prostaglandin synthetase inhibitors.

Author:

Littlewood J M,Lee M R,Meadow S R

Publisher

BMJ

Subject

Pediatrics, Perinatology and Child Health

Reference15 articles.

1. Genetic hypertension in rats is accompanied by a defect in renal prostaglandin catabolism;Armstrong, J.M.; Blackwell, G.J.; Flower, R.J.; McGiff, J.C.; Mullaine, K.M.; Vane, J.R.;Nature,1976

2. Hyperplasia of the juxtaglomerular complex with hyperaldosteronism and hypokalemic alkalosis;Bartter, F.C.; Pronove, P.; Gill, J.R.; MacCardle, R.C.;American Journal of Medicine,1962

3. Hyperaldosteronism, hyperplasia of the juxtaglomerular complex, normal blood pressure and dwarfism: report of a case;Bryan, G.T.; MacCardle, R.C.; Bartter, F.C.;Pediatrics,1966

4. Juxtaglomerular cell hyperplasia and secondary hyperaldosteronism (Bartter's syndrome): a re-evaluation of the pathophysiology;Cannon, J.; Leeming, J.M.; Sommers, S.C.; Winters, R.W.; Laragh, J.H.;Medicine,1968

5. Role of prostaglandins in the pathogenesis of Bartter's syndrome;Fichman, M.P.; Telfer, N.; Zia, P.; Speckart, P.; Golub, M.; Rude, R.;American Journal of Medicine,1976

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