1. The Veterans Administration Cooperative Study of Pulmonary Function sodium excretion is not due to a concomitant fall in atrial natriuretic peptide. Levels of atrial III. Mortality in relation to respiratory function in chronic obstructive pulmonary disease;Renzetti, A.D.; McClement, J.H.; Litt, B.D.;Am. Med; 41: 11529. natriuretic peptide are raised in cor pulmonale and increase further on hypertonic saline loading.7 Alternatively, the opposing effects of systemic and intrarenal angiotensin II might explain these findings. Intrarenal angiotensin II maintains the glomerular filtration rate via efferent arteriolar vasoconstriction,1966
2. Right ventricular function in cor pulmonale;Macnee, W.;Cardiology,1988
3. A study of cor pulmonale in patients with chronic bronchitis;Platts, M.M.; Hammond, J.D.S.; Stuart-Harris, C.H.;Q I Med
4. Abnormalities of sodium and water handling in chronic obstructive lung disease. Arch in angiotensin II might lower the glomerular filtration rate and would be expected to reduce sodium excretion. The lack of change in the glomerular filtration rate, despite systemic;Farber, M.O.; Roberts, L.R.; Weinberger, M.H.; Robertson, G.L.; Fineberg, N.S.; Manfredi, F.;Intern Med,1982
5. Mechanisms of hyponatremia and edema in chronic obstructive pulmonary disease: clinical significance;Farber, M.O.; Manfredi, F.;Pract Cardiol,1984