The cytokine response to critical illness.

Author:

Foex B A,Shelly M P

Publisher

BMJ

Subject

Critical Care and Intensive Care Medicine,General Medicine,Emergency Medicine

Reference57 articles.

1. Anti-cachectin/TNF monoclonal antibodies prevent septic shock during lethal bacteraemia;Tracey, K.J.; Fong, Y.; Hesse, D.G.; Manogue, K.R.; Lee, A.T.; Kuo, G.C.;Nature,1987

2. Antibodies to cachectin/tumor necrosis factor reduce interleukin 1 B and interleukin 6 appearance during lethal bacteremia;Fong, Y.; Tracey, K.J.; Moldower, L.L.; Hesse, D.G.; Manogue, K.B.; Kenney, J.S.;J Exp Med,1989

3. Experimental endotoxaemia in humans: analysis of cytokine release and coagulation, fibrinolytic and complement pathways;van Deventer SJH; HR, Buller; ten Cate JW; LA, Aarden; CE, Hack; A, Sherle;Blood,1990

4. Detection of circulating tumor necrosis factor after endotoxin administration;Michie, H.R.; Manogue, K.R.; Spriggs, D.R.; Reuhang, A.; O'Dwyer, S.O.; Dinarello, C.A.;N EngljMed,1988

5. Tumor necrosis factor (cachectin) is an endogenous pyrogen and induces production of interleukin 1;Dinarello, C.A.; Cannon, J.G.; Wolff, S.M.; Bernheim, H.A.; Beutler, B.; Cerami, A.;J Exp Med,1986

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