1. Prenatal diagnosis and carrier detection of Duchenne muscular dystrophy with closely reaction in older DMD patients (30 kd,34 1460,33 and this study for P20 (fig 3) and 1460 (fig 4 F, I, L)), nor linked RFLPs;Bakker, E.; Hofker, M.H.; Goor, N.;Lancet,1985
2. Prenatal diagnosis of Duchenne muscular dystrophy: a three year experience in a in mdx mice. However, we cannot exclude the sharing of antigenic determinants between putative, exclusively embryonic, dystrophin-like proteins with a similar appearance early in embryogenesis. To investigate this further, additional biochemical rapidly evolving field;Bakker, E.; Bonten, E.J.; Veenema, H.;J Inher Metab Dis,1989
3. Complete cloning of the Duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals;Koenig, M.; Hoffman, E.P.; C1, Bertelson; Monaco, A.P.; Feener, C.; Kunkel, L.M.;Cell,1987
4. The molecular basis for Duchenne versus Becker muscular dystrophy: correlation of severity with typeofdeletion;Koenig, M.; Beggs, A.H.; Moyer, M.;AmJ Hum Genet,1989
5. Dystrophin: the protein product of the Duchenne muscular dystrophy locus;Hoffman, E.P.; Brown, R.H.; Kunkel, L.M.;Cell,1987