Variation of phenotype in patients with glucocorticoid remediable aldosteronism.

Author:

Gates L J,MacConnachie A A,Lifton R P,Haites N E,Benjamin N

Publisher

BMJ

Subject

Genetics (clinical),Genetics

Reference8 articles.

1. Autosomal dominant transmission and absence of HLA linkage in dexamethasone suppressible hyperaldosteronism;New, M.I.; Oberfield, S.E.; Levine, L.S.; etal;Lancet,1980

2. Hypertension, increased aldosterone secretion and low plasma renin activity relieved by dexamethasone;Sutherland, D.J.A.; Ruse, J.L.; Laidlaw, J.C.;Can Med Assoc J,1966

3. A new form of congenital adrenal hyperplasia;New, M.I.; Peterson, R.E.;JClin EndocrinolMetab,1967

4. Miura K, Yoshinaga K, Goto K, etal. A case ofglucocorticoidresponsive aldosteronism. JClin EndocrinolMetab 1968;28: 1807-15.

5. A kindred with familial glucocorticoid-suppressible aldosteronism;Giebink, G.S.; Gotlin, R.W.; Biglieri, E.G.; Katz, F.H.;J Clin EndocrinolMetab,1973

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