Quantitation of infarct size in man by means of plasma enzyme levels.

Author:

Witteveen S A,Hemker H C,Hollaar L,Hermens W T

Publisher

BMJ

Subject

Cardiology and Cardiovascular Medicine

Reference13 articles.

1. Serum transaminase levels in experimental myocardial infarction;Agress, C.M.; Jacobs, H.I.; Glassner, H.F.; Lederer, M.A.; Clerk, W.G.; Wroblewski, F.; Karmen, A.; LaDue, J.S.,1955

2. The disappearance rate of glutamic oxaloacetic transaminase from the circulation and its distribution in the body's fluid compartments and secretions;Dunn, M.; Martin, J.; Reissmann, K.R.;Journal of Laboratory and Clinical Medicine,1958

3. Coronary artery reperfusion. II. Reduction of myocardial infarct size at one week after the coronary occlusion;Ginks, W.R.; Sybers, H.D.; Maroko, P.R.; Covell, J.W.; Sobel, B.E.; Ross, J.;Journal of Clinical Investigation,1972

4. Quantitative study of infarcted myocardium in cardiogenic shock;Harnarayan, C.; Bennett, M.A.; Pentecost, B.L.; Brewer, D.B.;British Heart Journal,1970

5. Effect of a thrombolytic agent (urokinase) on necrosis after acute myocardial infarction (AMI);Hermens, W.Th; Witteveen, S.A.G.J.; Hollaar, L.; Hemker, H.C.,1975

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