Is Helicobacter pylori associated hypergastrinaemia due to the bacterium's urease activity or the antral gastritis?

Author:

Chittajallu R S,Dorrian C A,Neithercut W D,Dahill S,McColl K E

Publisher

BMJ

Subject

Gastroenterology

Reference23 articles.

1. Amoxycillin and tinidazole for Campylobacter pylori gastritis in children: assessment by serum IgG antibody, pepsinogen I and gastrin levels;Oderda, G.; Vaira, D.; Holton, J.; Ainley, C.; Altare, F.; Ansaldi, N.;Lancet,1989

2. Antral Helicobacter pylori, hypergastrinaemia and duodenal ulcers: effect of eradicating the organism;Levi, S.; Beardshall, K.; Swift, I.; Foulkes, W.; Playford, R.; Ghosh, P.;BMJ,1989

3. Ablation of exaggerated meal stimulated gastrin release in duodenal ulcer patients after clearance of Helicobacter pylori infection;Graham, D.Y.; Opekun, A.; Lew, G.M.; Evans, D.J.; Klein, P.D.; Evans, D.G.;AmJ Gastroenterol,1990

4. Effect of increasing Helicobacter pylori ammonia production by urea infusion on plasma gastrin concentrations;Chittajallu, R.S.; Neithercut, W.D.; MacDonald, A.M.I.; McColl, K.E.L.;Gut,1991

5. Lowered gastrin and gastric acidity after eradication of Campylobacter pylori in duodenal ulcer;McColl, K.E.L.; Fullarton, G.M.; Nujumi, A.M.; Brown, MacDonald A.M.; IL, Hilditch; T.E.;Lancet,1989

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