1. Clinicopathological findings in patients with primary and secondary defects of neutrophil mobility;Farhoudi, A.; M, Harvey B.; F, Soothill J.;Arch Dis Child; other patients with neutrophil mobility defects7-9 suggest that ascorbic acid and levamisole act by way of an alteration of the,1978
2. Serum monoclonal immunoglobulins in childhood;Danon, F.; Seligmann, M.;Arch Dis Child,1973
3. Deficiency of kor xtype immunoglobulins. Blood 1976; 47: 79-89. ratios of cyclic-AMP to cyclic-GMP which in turn influence microtubule assembly.'0 The drug effect therefore is consistent with a possible underlying biochemical abnormality but;Barandun, S.; Morell, A.; Skvaril, F.; Oberdorfer, A.
4. Leukocyte locomotion and chemotaxis. New methods for evaluation and demonstration of a cell-derived chemotactic factor;H, Zigmund S.; G, Hirsch J.;J Exp Med,1973
5. A proposal of its nature. Neither levamisole nor ascorbic acid treatment was of therapeutic benefit in Case 8, although the lack of response may have been due to pre-existing lung damage. Both the clinical features and laboratory results in;U, Keller H.; C, Wilkinson P.; M, Abercrombie;Cases; and 10 are consistent with a primary defect of neutrophil mobility. The similar symptoms in Cases 3 and 4 suggest that they had the same disease. The case for for the definition of terms related to locomotion of leucocytes and other cells. Clin Exp Immunol,1977