Distribution of Helicobacter pylori colonisation and associated gastric inflammatory changes: difference between patients with duodenal and gastric ulcers.

Author:

Louw J A,Falck V,van Rensburg C,Zak J,Adams G,Marks I N

Publisher

BMJ

Subject

General Medicine,Pathology and Forensic Medicine

Reference17 articles.

1. Helicobacter pylori and peptic ulcers: the present position;Moss, S.; Calam, J.;Gut,1992

2. the most common bacterial infection in Africa: a random serological survey;Holcombe, C.; Omotara, B.A.; Eldridge, J.; pylori, Jones D.M.H.;Am J Gastroenterol,1992

3. Gastric glycerolipid as a receptor for Campylobacter pylori;Lingwood, C.A.; Pellizzari, A.; Law, H.; Sherman, P.; Drumm, B.;Lancet,1989

4. N-acetylneuraminallactose-binding fibrillar hemagglutinin of Campylobacter pylori: a putative colonization factor antigen;Evans, D.G.; Evans, D.J.; Moulds, J.J.; Graham, D.Y.;Infect Immun,1989

5. Campylobacter pylori and duodenal ulcers: the gastrin link;Levi, S.; Haddad, G.; Ghosh, P.; Beardshall, K.; Playford, R.; Calam, J.;Lancet,1989

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