1. Nitric oxide: physiology, pathophysiology and pharmacology;Moncada, S.; Palmer, R.; Higgs, E.;Pharmacol Rev,1991
2. Increased gene expression for VEGF and the VEGF receptors KDR/Flk and Flt in lungs exposed to acute or to chronic hypoxia. Modulation of gene expression by nitric oxide;Tuder, R.; Flook, B.; Voelkel, N.;J Clin Invest,1995
3. Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines;R, De Caterina; P, Libby; HB, Peng; VJ, Thannickal; TB, Rajavashisth; MA, Gimbrone;J Clin Invest,1995
4. Impairment of endotheliumdependent pulmonary artery relaxation in chronic obstructive lung disease;AT, Dinh Xuan; TW, Higenbottam; CA, Clelland; J, Pepke-zaba; G, Cremona; AY, Butt;N Engl _J Med,1991
5. Loss of endothelium-dependent relaxant activity in the pulmonary circulation of rats exposed to chronic hypoxia;Adnot, S.; Raffestin, B.; Eddahibi, S.; Braquet, P.; Chabrier, P.E.;JGClin Invest,1991