1. Prevention of exercise-induced bronchoconstriction by inhaled furosemide;Bianco, S.; Vaghi, A.; Robuschi, M.; Pasargiklian, M.;Lancet,1988
2. Frusemide antagonises exerciseepithelial cells and then from the submucosa. The action of frusemide may be in reducing the potential for the submucosa to become dehydrated by blocking the movement of water by its action on the Na+/K+/2Clcotransporter while permitting water loss from the epithelial cell in response to an osmotic stimulus. Protecting the submucosa from possible dehydration and consequent increases in osmolarity26 would decrease the potential for further mediator release from mast cells and cause C afferent fibres to fire27 and microvascular leakage.28 All these events induced but not histamine-induced bronchospasm. Aust NZJMed;Feather, I.R.; Olson, L.G.,1991
3. Effect of inhaled furosemide on the bronchial response to methacholine and cold-air hyperventilation challenges;Grubbe, R.E.; Hopp, R.; Dave, N.K.; Brennan, B.; Bewtra, A.; Townley, R.;J Allergy Clin Immunol,1990
4. Inhaled furosemide is highly effective in preventing ultrasonically nebulised water bronchoconstriction;Robuschi, M.; Gambaro, G.; Spagnotto, S.; Vaghi, A.; Bianco, S.;Pulmizoniary Pharnitacol,1989
5. Inhaled furosemide prevents both the bronchoconstriction and the increase in neutrophil Inhaled frisemide inihibits airway response to hyperosniolar saline chemotactic activity induced by ultrasonic "fog" of distilled water in asthmatics;Moscato, G.; Dellabianca, A.; Falagiani, P.; Mistrello, G.; Rossi, G.; Rampulla, C.;Ani Rev Respir Dis,1991