Transient rise in serum interleukin-8 concentration during acute myocardial infarction.

Author:

Abe Y,Kawakami M,Kuroki M,Yamamoto T,Fujii M,Kobayashi H,Yaginuma T,Kashii A,Saito M,Matsushima K

Publisher

BMJ

Subject

Cardiology and Cardiovascular Medicine

Reference21 articles.

1. Accumulation of polymorphonuclear leukocytes during 3-hour experimental myocardial ischemia;Rb, Engler; MD, Dahlgren; MA, Peterson; A, Dobbs; G, Schmid-Schoenbein;Am j Physiol,1986

2. Role of leukocytes in the response to acute myocardial ischemia and reflow in dogs;Engler, R.I.; Dahlgren, M.D.; Morris, D.; Peterson, M.A.; Schmid-Schoenbein, G.;Am Y Physiol,1986

3. Oxygen-mediated myocardial damage during ischemia and reperfusion: role of the cellular defense against oxygen toxicity;Ferrari, R.; Ceconi, C.; Curello, S.; Guarnieri, C.; Caldarera, C.M.; Albertini, A.;J Mol Cell Cardiol,1985

4. Direct measurement of free radical generation following reperfusion;Zweier, J.L.; Flaherty, J.T.; Weisfeldt, M.L.,1987

5. Inflammatory response, neutrophil activation, and free radical production after acute myocardial infarction: effect of thrombolytic treatment;Bell, D.; Jackson, M.; Nicoll, J.J.; Millar, A.; Dawes, J.; Muir, A.L.;Br Heart J,1990

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